Calcium Channel Modulation as an All-Purpose Mechanism for Short-Term Synaptic Plasticity
نویسندگان
چکیده
The paper by Mochida et al. in this issue of Neuron proposes that synaptic facilitation, depression, and augmentation all arise from calcium-dependent regulation of calcium channels in the presynaptic terminal. Their proposal provides a unifying explanation for several forms of short-term presynaptic plasticity.
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عنوان ژورنال:
- Neuron
دوره 57 شماره
صفحات -
تاریخ انتشار 2008